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Abstract

Neurological diseases account for the leading cause of disability and second leading cause of death, worldwide; however, treatments remain severely limited. Despite preclinical success, very few have been successfully translated for clinical use. Here, we discuss strategies to overcome this in two major categories: improved therapeutics and improved preclinical disease models. Perhaps an obvious – if not simple – approach is to develop improved therapeutics that more effectively treat neurological disorders. Recently, extracellular vesicles (EVs), nanoscale particles secreted by cells containing proteins and genetic material, have been recognized for their role in intercellular communication. As many benefits of cell therapies appear to be due to paracrine factors and signaling – known as the “bystander effect” – EVs appear to recapitulate some, if not most, of these effects, without associated drawbacks (e.g., tumorigenicity, ability to cross the blood-brain barrier). Here, we examine the therapeutic potential of neural stem cell-derived EVs to treat traumatic brain injury, demonstrating improved structural protection and functional recovery 4 weeks post-injury.Additionally, more physiologically relevant disease models and assays are needed to study neurological disorders and evaluate novel therapeutics. Electrophysiology is a key endpoint to assess neural health in vitro, and improved technologies (i.e., microelectrode arrays; MEAs) allow for higher throughput and detailed network analysis. While these are useful tools, data analysis can be complicated and hinder efficacy. We developed a novel index to improve interpretation of MEA and electrophysiological data by condensing high dimensional data into a single score, allowing culture health and effects of potential therapeutics to be assessed more effectively, significantly improving the applicability of MEAs to evaluate disease phenotypes and treatments. Finally, we discuss implementation of electrophysiology in brain organoids to bridge the gap between traditional in vitro and in vivo models. While organoids have provided considerable insight into neural development and pathologies, functional analysis is needed to reach their full modeling potential. We highlight and propose the application of novel electrophysiological methods to study brain organoids and their improvements over current methods. Together, these studies support several strategies to improve therapeutic development for neurological disorders, including both improved treatments and more effective modeling approaches and evaluation.

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